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News Release / Events
DHA helps prevent Alzheimer’s
Fish Oil Users, Rejoice: there’s even more evidence to support DHA’s role in
Alzheimer’s disease prevention. A recent study published in The Journal of
Neuroscience shows that eating a diet rich in DHA, or docosahexaenoic acid, can
limit build-up of beta-amyloid on the brain, a condition commonly seen in
Alzheimer’s patients (2005, vol.25, no.12). Scientists fed mice different diets
of omega-3 fatty acids to determine that a high DHA intake can reduce brain
plaque levels by about 40 percent. DHA is found naturally in fatty fish and
organ meats.
Press Release – Omega 3
A new study explains how a diet high in oily fish like salmon and mackerel
improves inflammatory conditions, particularly in combination with low doses of
aspirin. In a study in the March 7 issue of The Journal of Experimental
Medicine, Arita and colleagues identify an anti-inflammatory lipid in humans
that is derived from an essential fatty acid in fish oil.
Fatty fish contain large amounts of omega-3 fatty acids--diet-derived essential
fatty acids known to benefit patients with cardiovascular disease and
arthritis. This research group recently identified a new class of
aspirin-triggered bioactive lipids, called resolvins, the activity of which may
in part explain the beneficial effects of omega-3 fatty acids. Resolvins are
made from the omega-3 fatty acids by cellular enzymes and can reduce
inflammation in mice. The main bioactive component of this class of lipids was
identified in mice and named resolvin E1.
The researchers have now identified this lipid in plasma taken from volunteers
given omega-3 fatty acids and aspirin. Human resolvin E1, the authors show,
inhibits both the migration of inflammatory cells to sites of inflammation and
the turning on of other inflammatory cells.
This study also reveals a potential pitfall of COX-2 inhibitors, drugs designed
to block inflammation, which have been shown to have negative cardiovascular
side effects. COX-2 is involved in making resolvin E1 and the authors suggest
that inhibition of vascular COX-2 by these inhibitors might block the synthesis
of resolvin E1, which would eliminate an important anti-inflammatory pathway.
The experiment to prove this idea, however, has yet to be done.
2005-07-06
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